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APC/C(Cdh1) targets brain-specific kinase 2 (BRSK2) for degradation via the ubiquitin-proteasome pathway.

机译：apC / C（Cdh1）通过遍在蛋白 - 蛋白酶体途径靶向脑特异性激酶2（BRsK2）进行降解。

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Ruwei Li; Bo Wan; Jun Zhou; Yingli Wang; Ting Luo; Xiuting Gu; Fang Chen; Long Yu;
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年度 2012
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1. Jab1 interacts with brain-specific kinase 2 (BRSK2) and promotes its degradation in the ubiquitin-proteasome pathway [J] . ZhouJ., WanB., LiR., Biochemical and Biophysical Research Communications . 2012,第4期

机译：Jab1与大脑特异性激酶2（BRSK2）相互作用，并在遍在蛋白-蛋白酶体途径中促进其降解
2. Jab1 interacts with brain-specific kinase 2 (BRSK2) and promotes its degradation in the ubiquitin-proteasome pathway [J] . ZhouJ., WanB., LiR., Biochemical and Biophysical Research Communications . 2012,第4期

机译：Jab1与大脑特异性激酶2（BRSK2）相互作用，并在遍在蛋白-蛋白酶体途径中促进其降解
3. The adaptor molecule CIN85 regulates Syk tyrosine kinase level by activating the ubiquitin-proteasome degradation pathway. [J] . Peruzzi G, Molfetta R, Gasparrini F, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2007,第4期

机译：衔接子分子CIN85通过激活泛素-蛋白酶体降解途径来调节Syk酪氨酸激酶水平。
4. DYSTROPHIN INVOLVED IN THE ATROPHIC RESPONSE OF SLOW MUSCLES TO HINDLIMB UNLOADING VIA CONCOMITANT ACTIVATION OF TGF-BETA1/SMAD3 SIGNALING AND UBIQUITIN-PROTEASOME DEGRADATION IN MICE [C] . Hongju Liu, Peng Zhang, Wenjiong Li, International astronautical congress . 2014

机译：TGF-BETA1 / SMAD3信号转导和泛泛素-蛋白质降解对小鼠慢肌的萎缩反应中神经营养不良蛋白的影响
5. A Global Kinase and Phosphatase Interaction Network in the Budding Yeast Reveals Novel Effectors of the Target of Rapamycin (TOR) Pathway. [D] . Sharom, Jeffrey Roslan. 2011

机译：萌芽酵母中的全球激酶和磷酸酶相互作用网络揭示了雷帕霉素（TOR）途径的目标的新型效应子。
6. APC/CCdh1 Targets Brain-Specific Kinase 2 (BRSK2) for Degradation via the Ubiquitin-Proteasome Pathway [O] . Ruwei Li, Bo Wan, Jun Zhou, -1

机译：apC / CCdh1瞄准脑特异性激酶2（BRsK2）用于降解经由泛素 - 蛋白酶体途径
7. Degradation of the Met tyrosine kinase receptor by the ubiquitin-proteasome pathway. [O] . Jeffers, M, Taylor, G A, Weidner, K M, 1997

机译：遍在蛋白-蛋白酶体途径降解Met酪氨酸激酶受体。

APC/C(Cdh1) targets brain-specific kinase 2 (BRSK2) for degradation via the ubiquitin-proteasome pathway.

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